Labels:text | screenshot | black | black and white | font | document OCR: the In patients with mal ignant hypertensic necrosis elevated blood pressure leads to fibrino i turn, of the small vessels of the kidney; this, us further stimulates excessive renin release and n of the exacerbates the hypertension by activat uch renin-angiotensin-aldosterone system. patients therefore have secondary hyperaldosteronism (Fig. 10.10). See also: · Conn's Syndrome
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